Lypolysis Explained!

What causes our body to burn fat?

When our bodies produce catecholamines, growth hormone, glucagon and testosterone we initiate the release of Camp (cyclic adenosine monophosphate) through enzymic reaction of ATP and the hormone signal in question. Through activation of Camp we then activate protein kinase A which triggers the activation of lipase. Lipase aids fat breakdown and as such triggers fat loss.

Now in the liver the activation of Camp is greatly effected by the release of glucagon, whilst in the muscle this activation is mediated through catecholamine. Although there is a cross-link between two catalysts.

We now have an environment in which to play with fat loss. As most of our body will process triglycerides into stored adipose tissue via assimilation in the liver we can control the amount of lypogenesis formed through managing glucagon. How we do this is by sustaining blood glucose levels at a low control through moderate fat intake, and low carb intake.

During training the increase awareness of catecholamines is seen on a low carbohydrate diet this is due to the increased perception or response the body has on fat releasing hormones noted during low carbohydrate diets. This is great for catecholamine activation of Camp toward lypolysis fueling muscle contraction.

However it must also be noted however that the body will convert available glycerol into usable glycogen for the energy need through the production of ketones. A prolonged exposure to ketongenic diets however will slow down thyroid regulation to cope with the oxaloacetate depletion and energy stored required to generate oxaloacetate due to low carbohydrate intake. Therefore increasing carbohydrate load periodically will provide the wood for the flame to keep burning whilst not disrupting biorhythm of fat releasing hormones over the fat loss period as a whole.

So this is why low carbohydrate diets work well for fat loss. Just remember however in the case of performance the abundance of available oxaloacetate will dictate the amount of ATP generation through mitochondrial activation of ATP via glycolysis, glucolysis and gluconeogensis. This is why when maximal performance is due one should have a healthy composition to utilise blood glucose efficiently through the metabolic load or need of activity. A fat athlete does not stand a chance of optimal performance as energy use will not be as great as that athlete could possibly produce.

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